An altered peptide ligand mediates immune deviation and prevents autoimmune encephalomyelitis
LB Nicholson, JM Greer, RA Sobel, MB Lees… - Immunity, 1995 - Elsevier
Abstract In experimental autoimmune encephalomyelitis (EAE) induced with myelin
proteolipid protein (PLP) peptide 139–151, we have previously shown that the disease is
mediated by Th1 cells, which recognize tryptophan 144 as the primary TCR contact point.
proteolipid protein (PLP) peptide 139–151, we have previously shown that the disease is
mediated by Th1 cells, which recognize tryptophan 144 as the primary TCR contact point.
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