[HTML] Axonal transport and secretion of fibrillar forms of α-synuclein, Aβ42 peptide and HTTExon 1
M Brahic, L Bousset, G Bieri, R Melki, AD Gitler - Acta neuropathologica, 2016 - Springer
Accruing evidence suggests that prion-like behavior of fibrillar forms of α-synuclein, β-
amyloid peptide and mutant huntingtin are responsible for the spread of the lesions that
characterize Parkinson disease, Alzheimer disease and Huntington disease, respectively. It …
amyloid peptide and mutant huntingtin are responsible for the spread of the lesions that
characterize Parkinson disease, Alzheimer disease and Huntington disease, respectively. It …
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