ER stress stimulates production of the key antimicrobial peptide, cathelicidin, by forming a previously unidentified intracellular S1P signaling complex

ER stress stimulates production of the key antimicrobial peptide, cathelicidin, by forming a previously unidentified intracellular S1P signaling complex

K Park, H Ikushiro, HS Seo, KO Shin… - Proceedings of the …, 2016 - National Acad Sciences

We recently identified a previously unidentified sphingosine-1-phosphate (S1P) signaling
mechanism that stimulates production of a key innate immune element, cathelicidin
antimicrobial peptide (CAMP), in mammalian cells exposed to external perturbations, such …

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